Autor(en):
Desai NR*, Kesari Kavindra K, Agarwal A
* Center for Reproductive Medicine, Glickman Urological and Kidney Institute and Obstetrics and Gynecology and Women's Health Institute, Cleveland Clinic, Cleveland, Ohio.
USA
Veröffentlicht in:
Reproductive Biology and Endocrinology 2009 Aug 13, 7:114
Veröffentlicht: 22.10.2009
auf EMF:data seit 21.05.2017
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Pathophysiologie der Mobiltelefon-Strahlung: oxidativer Stress und Karzinogenese mit Schwerpunkt auf dem männlichen Fortpflanzungssystem.

Pathophysiology of cell phone radiation: oxidative stress and carcinogenesis with focus on male reproductive system.

Original AbstractÜbersetzung n.n. vorhanden!

Hazardous health effects stemming from exposure to radiofrequency electromagnetic waves (RF-EMW) emitted from cell phones have been reported in the literature. However, the cellular target of RF-EMW is still controversial. This review identifies the plasma membrane as a target of RF-EMW. In addition, the effects of RF-EMW on plasma membrane structures (i.e. NADH oxidase, phosphatidylserine, ornithine decarboxylase) and voltage-gated calcium channels are discussed. We explore the disturbance in reactive oxygen species (ROS) metabolism caused by RF-EMW and delineate NADH oxidase mediated ROS formation as playing a central role in oxidative stress (OS) due to cell phone radiation (with a focus on the male reproductive system). This review also addresses: 1) the controversial effects of RF-EMW on mammalian cells and sperm DNA as well as its effect on apoptosis, 2) epidemiological, in vivo animal and in vitro studies on the effect of RF-EMW on male reproductive system, and 3) finally, exposure assessment and dosimetry by computational biomodeling.

Exposition:

Mobiltelefone

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Zusammenfassung

Auszug aus Original-Abstract: [...] Commercially available cellular phones might affect cell function via non-thermal effects. [...] Prolonged exposure to RF-EMW can also cause DNA damage (by prolonged OS), which may accelerates neuronal and spermatozoal cell death and promote neurodegenerative processes as well as promote brain and testicular carcinogenesis. Any tumor promoting effects of RF-EMW might be due to the effect it has on PKC, ODC, intra cellular calcium spikes and stimulation of stress kinase. [...] OS and changes in PKC activity might lead to the RF-EMW related infertility observed in numerous studies.

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