Tillmann T*, Ernst H, Streckert J, Zhou Y, Taugner F, Hansen V, Dasenbrock C.
* Fraunhofer Institute for Toxicology and Experimental Medicine, Hannover, Germany.
Published in:
Int J Radiat Biol 2010; 86 (7): 529-541
Published: July 2010
on EMF:data since 20.02.2018
Further publications: Studie gefördert durch:

Stiftung (Grant number: 11581/23718); Projektbegleitung: Dr. Lorenzo Tomatis (†, Trieste, Italien) und Prof. Ulrich Mohr (Hannover, Deutschland).

Keywords for this study:
Growth of cancer cells, tumor promotion
Medical/biological studies

Indication of cocarcinogenic potential of chronic UMTS-modulated radiofrequency exposure in an ethylnitrosourea mouse model.

Original Abstract

Purpose: To evaluate putative effects on tumour susceptibility in mice exposed to a UMTS (universal mobile telecommunications system) test signal for up to 24 months, commencing with embryo-fetal exposure.

Material and methods: Animals were exposed to UMTS fields with intensities of 0, 4.8, and 48 W/m²), the low-dose group (4.8 W/m²) was subjected to additional prenatal ethylnitrosourea treatment (40 mg ENU/kg body weight).

Results: The high-level UMTS exposure (48 W/m²), the sham exposure, and the cage control groups showed comparable tumour incidences in the protocol organs. In contrast, the ENU-treated group UMTS-exposed at 4.8 W/m²) displayed an enhanced lung tumour rate and an increased incidence of lung carcinomas as compared to the controls treated with ENU only. Furthermore, tumour multiplicity of the lung carcinomas was increased and the number of metastasising lung tumours was doubled in the ENU/UMTS group as compared to the ENU control group.

Conclusion: This pilot study indicates a cocarcinogenic effect of lifelong UMTS exposure (4.8 W/m²) in female B6C3F1 descendants subjected to pretreatment with ethylnitrosourea.


Ethylnitrosourea | ENU | UMTS | electromagnetic fields | cellular phone | health effects | cancer | B6C3F1 mice

Source: PubMed

1900-2100 MHz
4,8; 48 W/m²